![]() ![]() The majority (86%) was in New York Heart Association (NYHA) class I, and 14% remained in NYHA class II. Univariate and multivariate analyses identified no tested variables as risk factors for reoperation. Actuarial freedom from any type of aortic reoperation or intervention was 86% at 1 year, 69% at 5 years, and 60% at 10 and 15 years. Risk factors for increased mortality by univariate analysis were as follows: (1) primary aortic anastomosis ( p = 0.03), (2) presence of complex anomalies ( p = 0.05), and (3) initial IAA repair performed before 1994 ( p = 0.05). There was an 81% 15-year survival for children in group I compared with a 54% for children in group II ( p ≪ 0.001). The actuarial survival including early mortality was 92% at 1 year, 81% at 5 years, and 76% at 10 and 15 years. Results: There were 5 early and 10 late deaths. Method of staged repair of IAA was to transect and turn down the left carotid artery and anastomosis it to the descending aorta ( n = 41) or graft interposition ( n = 2) combined with a pulmonary artery (PA) banding followed in a few months by delayed ventricular septal defect (VSD) closure and PA de-banding. Group II ( n = 14) were patients with Taussig–Bing double outlet right ventricle ( n = 6) or truncus arteriosus ( n = 8). Non-complex patients (group I, n = 51) had a ventricular septal defect (87%), aortopulmonary window (8%), and left ventricular outflow tract obstruction (27%). The surgical management strategy at our institution between 19 has been one-stage complete repair ( n = 13) or staged repair ( n = 52) in selected patients. Methods: Sixty-five patients (mean age, 16.9 ± 41.7 days) were diagnosed with IAA and referred for surgical therapy. We report our results for staged repair of this complex congenital malformation. Objective: The surgical results for the repair of interrupted aortic arch (IAA) have evolved in recent years. ![]()
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